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In contrast, DFP treatment significantly decreased Fe 2+ levels in optic nerve tissue, downregulated TFR1 and Iba-1 expression, and inhibited iron deposition, ferroptosis, microglial activation, and astrocyte proliferation, thereby reducing myelin loss and optic nerve damage in MS mice ( In summary, MAT, Dabrafenib, bone marrow mesenchymal stem cell-derived exosomes miR-367-3p, and DFP modulate ferroptosis, iron deposition, lipid peroxidation, neuroinflammation, microglial activation, and myelin loss, thereby attenuating MS-related pathological damage

The molecular genetic basis of age-related macular degeneration: an overview

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