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Guided by the glucose metabolic cascade, we comprehensively discuss nano-enabled strategies to achieve antitumor efficacy through inhibiting glucose transportation, blocking glucose supply, accelerating glucose depletion, suppressing glycolytic enzymes, and concurrently inhibiting oxidative phosphorylation

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Keywords : copper, cuproptosis, copper homeostasis, cardiovascular disease, mitochondrial dysfunction Cell death is a critical driver of cardiovascular disease pathogenesis, contributing to tissue damage, dysfunction, and inflammation [1]

But 6-8 weeks of consistent use can lead to impressive changes in skin and hair appearance, and the GHK-Cu safety profile is high