In SA-deficient NahG plants, however, the induction of NtPR-1a transcripts was delayed as it was first detectable following a 24 h SA exposure, while 48 h of both SA and OTC treatments resulted in further elevated expression of NtPR-1a ( Figure S3 )
2004;8:S3741
Water, Triethylhexanoin, Hydrogenated Poly(C6-14 Olefin), Butylene Glycol, Niacinamide, Methylpropanediol, 1,2-Hexanediol, Phenyl Trimethicone, Diethoxyethyl Succinate, Polyglyceryl-10 Myristate, Borago Officinalis Extract, Centaurea Cyanus Flower Extract, Chamomilla Recutita (Matricaria) Flower/Leaf Extract, Lavandula Angustifolia (Lavender) Flower Water, Hyacinthus Orientalis (Hyacinth) Extract, Salvia Sclarea (Clary) Extract, Ethylhexylglycerin, Hibiscus Esculentus Fruit Extract, Corchorus Olitorius Leaf Extract, Carum Petroselinum (Parsley) Extract, Hexyl Cinnamal, Limonene, Linalool, Fragrance, Hippophae Rhamnoides Oil, Allantoin, Hydrogenated Lecithin, Sodium Gluconate, Glutathione, Adenosine, Cellulose Gum, Melia Azadirachta Leaf Extract, Melia Azadirachta Flower Extract, Curcuma Longa (Turmeric) Root Extract, Ocimum Sanctum Leaf Extract, Corallina Officinalis Extract, Biotin, Biotinoyl Tripeptide-1, Acetyl Hexapeptide-8, Tripeptide-1, Tripeptide-3, Myristoyl Pentapeptide-17, Hexapeptide-9, Gluconolactone, Alpha-Arbutin, Thioctic Acid
The free tripeptide was first identified in human plasma in 1973 by Loren Pickart as a factor supporting cell proliferation in liver cell cultures, and the copper-binding role of the peptide was recognized shortly afterward as the molecular basis for many of the activities attributed to GHK
