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This figure provides a detailed analysis of these pathways and highlights potential therapeutic targets such as GPX4, System Xc-, NRF2, ACSL4, Coenzyme Q10, and FSP1

Since multiple inflammatory mediators are secreted within the lung microenvironment to drive fibrotic remodeling in asthma, we further investigated whether IL-24 stimulation could induce airway epithelial cells to produce pro-inflammatory or profibrotic factors

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Research Use Only The following peer-reviewed publications reference compounds for laboratory and in vitro research purposes only